Benjamin Elsbernd, M.D. Assistant Professor School Medical School Department Internal Medicine Biography Benjamin Elsbernd, M.D., is an Associate Professor in the Department of Internal Medicine at UT Southwestern Medical Center and a member of the Division of Digestive and Liver Diseases. Raised in Fort Worth, Dr. Elsbernd holds a bachelor’s degree in cellular and molecular biology from the University of Texas at Austin. He received his medical degree from UT Southwestern, where he also completed his internal medicine residency training and a fellowship in gastroenterology. He received further training as a Fellow in the American Neurogastroenterology and Motility Society Clinical Training Program at Washington University in St. Louis. Certified by the American Board of Internal Medicine, he joined the UT Southwestern Faculty in 2020. Dr. Elsbernd’s research investigations have resulted in several publications in peer-reviewed journals. At UT Southwestern, Dr. Elsbernd serves as the Associate Program Director for the Gastroenterology and Hepatology Fellowship, and as the Dallas VA Fellowship Site Director. Dr. Elsbernd is active in several professional societies, including the American College of Gastroenterologists, the American Gastroenterological Association, and the American Neurogastroenterology and Motility Society. Dr. Elsbernd’s clinical interests include esophageal diseases including gastroesophageal reflux, gastrointestinal motility disorders, esophageal function testing, and Barrett’s esophagus. Personal Note Dr. Elsbernd’s interests outside of medicine include cooking, running, reading and spending time with his family. Research Interest Barrett's Esophagus Esophageal Function Testing Esophageal Motility Disorders Gastroesophageal Reflux Disease Medical Education Publications Featured Publications Gastric residual volume after split-dose compared with evening-before polyethylene glycol bowel preparation. Agrawal D, Elsbernd B, Singal A, Rockey D Gastrointest. Endosc. 2015 Sep Profound hypoglycemia in starved, ghrelin-deficient mice is caused by decreased gluconeogenesis and reversed by lactate or fatty acids. Li RL, Sherbet DP, Elsbernd BL, Goldstein JL, Brown MS, Zhao TJ, J. Biol. Chem. 2012 May 287 22 17942-50 Results 1-2 of 2 1 Professional Associations/Affiliations American Gastroenterological Association (2015) American Neurogastroenterology and Motility Society (2018)
